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Report #2905307

Received Jul 30, 2026

HospitalizedER / ED visitOffice visitDisabled
A note on interpretation. VAERS reports are unverified and may be incomplete or coincidental. A report does not establish that a vaccine caused an event, and counts should not be used to calculate incidence or infer causation. Full disclaimer

Overview

Sex
Female
Age
36 yrs
State
MI
Recovered
Not recovered
Vaccinated
Oct 26, 2021
Onset
Sep 1, 2024
Days to onset
1041
Hospital days
5

Vaccines (3)

TypeNameManufacturerDoseLotRoute / Site
COVID19COVID19 (COVID19 (NOVAVAX))NOVAVAX1EN5318SYR / LA
COVID19COVID19 (COVID19 (PFIZER-BIONTECH))PFIZER\BIONTECH2EN6198SYR / LA
COVID19COVID19 (COVID19 (PFIZER-BIONTECH))PFIZER\BIONTECH3FC3181SYR / LA

Symptoms (33)

Angiotensin converting enzyme increasedAntinuclear antibodyBalance disorderBlood albumin increasedBlood gasesBlood lactic acid increasedCOVID-19Capillary leak syndromeComplement factor C3 increasedComputerised tomogram thorax abnormalDizzinessDyspnoeaExercise tolerance decreasedFatigueHeadacheHyperventilationHypovolaemiaLactic acidosisLoss of personal independence in daily activitiesMalaiseMetabolic function test abnormalMicroangiopathyNauseaOvarian cystPresyncopePulmonary function test abnormalPyrexiaRespiratory alkalosisRespiratory symptomRheumatoid factorUltrasound pelvis abnormalUpper respiratory tract infectionWeight fluctuation

Symptom narrative

Following sequential COVID-19 infections and COVID-19 vaccination, I experienced progressive, severe systemic microvascular and inflammatory symptoms. My infection history includes documented COVID-19 cases on 12/02/2021, 11/13/2022, 12/21/2023, and 01/24/2024. Each infection presented with worsening severity, including fevers reaching 105°F unresponsive to Tylenol and Motrin, severe headache, and profound fatigue. Subsequent to these events, I developed a severe 6-week non-COVID upper respiratory infection that failed to resolve until treated with high-dose oral steroids. Following steroid cessation, my symptoms progressed into chronic, multi-systemic microvascular dysfunction. Key Clinical Features & Symptoms: Systemic Capillary Leak & Fluid Shifts: Recurrent, cyclic weight fluctuations of 12 to 14 lbs within 48?72 hour windows (e.g., 147 lbs down to 142.9 lbs). Fluid shifts are accompanied by acute intravascular volume depletion, severe pre-syncope/dizziness, unsteadiness, headache, and nausea. Post-Exertional Anaerobic Stress: Marked post-exertional malaise and exercise intolerance following minor daily exertion. Pulmonary & Vascular Symptoms: Air hunger, breathing changes, and exertion-triggered vascular exhaustion - used to be a collegiate runner that was still active/competitive now can no longer run.

Current illness

Covid 12-2-2021

Medical history

none known before this except monitoring a bicuspid aortic valve

Other medications

Cholestoff Complete, occasional zyrtec use

Allergies

Statins, cat

Lab data

Endothelial, Inflammatory & Immune Markers:Angiotensin-Converting Enzyme (ACE) Trend (Serially Escalating):April 2025: 41 U/L (Baseline)July 2025: 52 U/L (Elevated)July 14, 2026: 75 U/L (Markedly elevated; confirms progressive microvascular endothelial inflammation)Complement System:Elevated Complement C3 levels documented, indicating active innate immune system / complement pathway activation.Autoimmune & Serological Panels:Autoimmune screening panels (including standard ANA, rheumatoid/connective tissue serologies) have remained seronegative/flat, pointing toward a non-classical, innate autoinflammatory or microvascular endotheliitis process rather than structural autoimmune joint disease.2. Metabolic & Arterial Blood Gas Trends (Recurrent Episodes):Lactic Acidosis (Recurrent / Post-Exertional Spikes):Documented resting lactic acidosis post-exertion, peaking at 3.7 mmol/L (Normal: 0.5?1.8 mmol/L) measured 5 days post-cardiopulmonary exercise testing (CPET). Recurrent episodes of elevated lactate reflect persistent microvascular tissue hypoxia and anaerobic cellular stress following minor physical strain.Respiratory Alkalosis (Recurrent / Compensatory):Multiple arterial blood gas (ABG) panels demonstrating recurrent respiratory alkalosis. This reflects compensatory hyperventilation ("air hunger") driven by metabolic lactic acidosis and impaired pulmonary capillary gas exchange.Intravascular Hemoconcentration:Serum Albumin peaking at 5.0 g/dL during acute fluid shift episodes, indicating severe plasma volume loss out of the blood vessels and into peripheral tissue spaces (third-spacing).3. Pulmonary & Diagnostic Imaging:Pulmonary Function Tests (PFTs): Demonstrated an isolated reduction in pulmonary diffusion capacity ($DLCO$), indicating a diffusion barrier across the alveolar-capillary membrane.Chest CT Scans (Jan 2025, May 2026, July 27, 2026): Consistently demonstrate stable 3?4 mm noncalcified pulmonary nodules (bilateral) and stable biapical/pleural-based scarring/fibrotic changes. Stable 1 cm posterior pleural-based thickening noted.Pelvic/Gynecological Ultrasound: Confirmed functional ovarian cysts (follicular/corpus luteum type); currently managed with Slynd (drospirenone) for ovulation suppression and hormonal/fluid stabilization.